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Mouse Estradiol (E2) ELISA Kit-Competitive

Product Overview

BioVenic Mouse Estradiol (E2) ELISA Kit-Competitive is designed for the quantitative determination of Mouse E2 in serum, plasma, tissue homogenate, cell culture supernatant, cell lysate, and other biological fluids using a Competitive ELISA method. For research use only.

Specifications

Assay Type ELISA-Competitive

Specificity The assay kit is specific for Mouse Estradiol (E2).

Target Species Mouse

Species Reactivity Mouse

Detection Range 25-1600 pg/mL

Reproducibility Intra-Assay: CV < 10%; Inter-Assay: CV < 12%

Assay Time Around 90 min

Sample Requirement Serum, plasma, tissue homogenate, cell culture supernatant, cell lysate, and other biological fluids.

Target Information

Estradiol is a hormone naturally produced by the ovaries in the human body. It plays a vital role in regulating the menstrual cycle and influences the cardiovascular, neurologic, skeletal, and vascular systems, among others. Research has demonstrated that estradiol enhances cortical and trabecular bone accrual and strengthens bones in an adolescent male-to-female mouse model undergoing gender-affirming hormone therapy. In a postmenopausal mouse model, estradiol treatment significantly increased bone mineral density, suggesting a potential protective role for estradiol against osteoporosis.

Target/Biomarker Mouse E2

Target Synonym 17B-Estradiol; Oestradiol; Beta-Estradiol

Shipping and Storage

This product is shipped with gel ice packs. It is recommended to store at 2-8 °C (Up to 6 months).

Documents

Datasheet

COA

To request a Certificate of Analysis, please enter the Lot No. in the search box. Note: Certificate of Analysis not available for kits.

The product is for research use only.
Not for commercial, prophylactic, diagnostic, or therapeutic applications.

References

Nie, Tian et al. "Estradiol increases cortical and trabecular bone accrual and bone strength in an adolescent male-to-female mouse model of gender-affirming hormone therapy." Bone research vol. 12,1 1. 11 Jan. 2024.